The antiinflammatory action mechanism of an antihomotoxic composita remedy
نویسندگان
چکیده
Traumeel, a combination homoeopathic preparation, has been used successfully for the treatment of trauma, inflammation and degenerative processes for more than 30 years (review in [11]). It has been shown in experimental investigations, primarily in ex-vivo studies in whole-blood cultures to which the individual components of the preparation had been added, that this homoeopathic antihomotoxic remedy stimulates the release of antiinflammatory cytokines, and particularly the inflammation-inhibiting “lead cytokine”, transforming growth factor beta (TGF-β), from leucocytes [6, 12, 13]. This gave rise to the theory that the antiinflammatory action of certain antihomotoxic remedies could be based on an “immunological bystander reaction” [13]. In this process, depending on the very low concentrations of active substances in the preparation (plant and mineral extracts, D2-D8), a TGF-β-producing type of lymphocyte is evidently formed (Th3 cells) which regulates immunological tolerance. This type of cell was first described by the Weiner study group after the administration of minute quantities of autologous antigens (in the lowest microgram range per unit of body weight) in autoimmune diseases, and was christened “bystander suppression” [22, 23]. The term immunological bystander reaction was introduced into the biological and medical literature by Heine in 1997 [9].
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